Restoring crashed estrogen on Aromasin

[verylargeshoes]

My sensitive estradiol test finally came back and I'm at 15 pg/mL. I have some symptoms that could be explained by low E2, so it looks like I overcorrected a bit with my use of Aromasin. I'll adjust dosage going forward, live and learn, no big deal. I'll be suboptimal for a couple of weeks.

I've read that some keep Dbol on hand to recover from crashed estrogen. Two questions come to mind now that I'm here for the first time, and I can't quite make sense of that idea. Would really appreciate insight from those who recommend or understand the thinking behind this practice.

1. Why would Dianabol be better at boosting estradiol levels than just injecting more testosterone? Is this because it has a significantly higher conversion rate, so you get more estradiol production per unit of other anabolic/androgenic effects? I don't know enough about this process to usefully reason about it, though I will definitely do some reading on it.

2. If I've so thoroughly depleted my aromatase enzymes that I can't aromatize enough test, what's left to convert dianabol into estradiol? Can a given residual concentration of aromatase enzymes produce more estradiol with dianabol present than with just test present?

The options I understand are A) reduce/discontinue aromasin and wait a week or two for the existing drug to clear out and for my body to then produce and use new enzymes on whatever aromatizable compounds are present, whether that's test or Dbol or MENT or whatever, or B) introduce exogenous estradiol.

 

[Bandern]

I’m not an expert but from what I understand dbol raises e2 pretty much instantly where test would take significantly longer. Plus you get to take dbol.

May be a reckless approach, it’ll take you from low e2 to high e2 pretty quick.

Curious what others will have to say, as I just got some dbol to have on hand for this reason (and for occasional pre workout for festivities)

 

[Photon]

My sensitive E2 on TRT was around 4 pg/mL so I'd be curious as well.
I'm blasting right and have Trest D added to keep my E2 up.

Dbol and MENT both produce a stronger version of E2 (methyl e2), AFAIK both do not turn up on labwork so it'll be much harder to dial in vs estrogenous e2 / test.

Another way to keep E2 up is HCG.

I think @Sampei might be running an experiment with injecting E2.

 

[Liter O' Test]

1. dbol is oral and works immediately and is a more potent form of estrogen
you havent depleted your aromatase enzymes

inject estrogen because you crashed yourself? man you are over thinking all of this

you didnt even say your dosages of test/asin

also 15 is not even crashed especially if you havent been at this level for awhile

 

[verylargeshoes]

1. dbol is oral and works immediately and is a more potent form of estrogen
you havent depleted your aromatase enzymes

inject estrogen because you crashed yourself? man you are over thinking all of this

you didnt even say your dosages of test/asin

Thanks for responding. Just to be clear, I'm not asking for help with what to do for myself, so my dosages don't matter. I'm asking about the specific mechanisms of the dianabol approach people have described so I can learn.

It seems you're saying that since dbol aromatizes to methylestradiol, even if the rate of aromatization isn't higher, the estrogenic effects are greater? And that its availability is more instant vs., say, test cyp or whatever the people who might use Dbol for this would have available. Both of those would definitely be advantages I hadn't considered. I was thinking of how TNE would perform vs. Dbol, so de-esterification wouldn't have been a bottleneck.

As for over-thinking -- it's interesting stuff. Thinking about it is fun.

 

[Photon]

Thanks for responding. Just to be clear, I'm not asking for help with what to do for myself, so my dosages don't matter. I'm asking about the specific mechanisms of the dianabol approach people have described so I can learn.

It seems you're saying that since dbol aromatizes to methylestradiol, even if the rate of aromatization isn't higher, the estrogenic effects are greater? And that its availability is more instant vs., say, test cyp or whatever the people who might use Dbol for this would have available. Both of those would definitely be advantages I hadn't considered. I was thinking of how TNE would perform vs. Dbol, so de-esterification wouldn't have been a bottleneck.

As for over-thinking -- it's interesting stuff. Thinking about it is fun.

TNE is kind of weird.
People using TNE purely for TRT (3x daily) seem to have stable in range e2 levels. However people using it for PWO doesn't seem to get an increase in E2.

 

[verylargeshoes]

TNE is kind of weird.
People using TNE purely for TRT (3x daily) seem to have stable in range e2 levels. However people using it for PWO doesn't seem to get an increase in E2.

WTF

 

[verylargeshoes]

Dbol and MENT both produce a stronger version of E2 (methyl e2), AFAIK both do not turn up on labwork so it'll be much harder to dial in vs estrogenous e2 / test.

Do you have a good reference on the properties of methyl estradiol? I am finding conflicting information, specifically about its affinity and activity at the estrogen receptor. The consistently stated difference between it and regular ol' 17beta estradiol appears to be greater resistance to clearance by the liver.

Another way to keep E2 up is HCG.

As I understand it, HCG stimulates the testicles to produce testosterone. Wouldn't that test aromatize at more or less the same rate as exogenous test?

 

[Tdeck81]

As I understand it, HCG stimulates the testicles to produce testosterone. Wouldn't that test aromatize at more or less the same rate as exogenous test?
[/QUOTE]

I posted a few videos on my baseline estradiol thread that touches on this and a few related issues. Might be worth a look when you get a few minutes. Some of the videos are kind of long

 

[Photon]

Do you have a good reference on the properties of methyl estradiol? I am finding conflicting information, specifically about its affinity and activity at the estrogen receptor. The consistently stated difference between it and regular ol' 17beta estradiol appears to be greater resistance to clearance by the liver.

Nope, i would be interested too

 

[verylargeshoes]

also 15 is not even crashed especially if you havent been at this level for awhile

Okay, not "crashed", but low enough that I am starting to demonstrate the symptoms of low E2. Without pharmaceutical intervention, I hover in the mid-20's, and when on higher doses of test, still feel good in the 30s to 40s. For example, I've got achy joints, and I never have achy joints.

 

[Sampei]

My sensitive E2 on TRT was around 4 pg/mL so I'd be curious as well.
I'm blasting right and have Trest D added to keep my E2 up.

Dbol and MENT both produce a stronger version of E2 (methyl e2), AFAIK both do not turn up on labwork so it'll be much harder to dial in vs estrogenous e2 / test.

Another way to keep E2 up is HCG.

I think @Sampei might be running an experiment with injecting E2.

Yes but need to wait autumn time, I'm on cruise mode right now not touching anything that is not a low dose of test. Need to detox myself

 

[newguy2]

Okay, not "crashed", but low enough that I am starting to demonstrate the symptoms of low E2. Without pharmaceutical intervention, I hover in the mid-20's, and when on higher doses of test, still feel good in the 30s to 40s. For example, I've got achy joints, and I never have achy joints.

I had this happen and did a shot of injectable dbol two days in a row. Seemed to bounce back I was having fatigue and some ed with it.

But dang I'm still limping from the dbol, never again.