MESO-Rx
Anabolic Steroids
Cardiac MRI Results (need help understanding)
- Thread starter benjaminnetenyahu
- Start date
trtchin
Member
To OP maybe I missed this but what do you consider to be a "just TRT" dose.
Def run as low as u can to feel good. IE put your levels at 800 instead of 1200
Def run as low as u can to feel good. IE put your levels at 800 instead of 1200
sakuraba126
Member
Post any ecgs you have, maybe even try pmcardio. It's ai, but Ai trained in ecg analysis by Dr Smith, as in smith-sgarbossa. It's a bit like reading tea leaves, but with the rest of your clinical picture may give some insight.
The issue with aas is it causes left ventricle muscle to grow. Excessively to the point the wall is so thick there isn't enough space for blood to fill before ejection.... Hence hf reduced Ef, vs hf preserved Ef, which indicates Rv dysfunction - percent of blood ejected, is normal, just the total amount the Rv pumps to lungs, lay, lv is less....
Hypokinesis is your lv not squeezing at full strength. Weird for hypertrophic obstructive cardiomyopathy, extra heart muscle should be hypeekinetic. The ra dilation (if I remember right) + the racing heart episode suggests SVT, possibly afib rvr. An electrical problem, but major stroke, heart attack risk in an aas user likely with high BP, high ldl.
The edema mentioned is concerning for an infectious issue, peri/myo carditis.
Any reduction in Ef =reduced blood through the coronary arteries, reduced O2 to the muscle makes cardiac tissue irritable and prone to electrical dysfunction like arrhythmias mentioned.
Npp/deca is notorious for lvh, add dnp induced tachycardia, maybe pericarditis from a injection infection and your SVT episode, weakened lv squeeze would track.
Your ecg may not be diagnostic but can show subtle signs of lvh (qrs amplitude v3-v6) pericarditis(low qrs amplitude, global ste, slanted tp) , hocm(dagger q) , arvdh(terminal qrs slur) , 1 AV block often precedes afib.a small infarct that killed some lv causing Hypokinesis, , bbbs, electrolyte derangement....
The issue with aas is it causes left ventricle muscle to grow. Excessively to the point the wall is so thick there isn't enough space for blood to fill before ejection.... Hence hf reduced Ef, vs hf preserved Ef, which indicates Rv dysfunction - percent of blood ejected, is normal, just the total amount the Rv pumps to lungs, lay, lv is less....
Hypokinesis is your lv not squeezing at full strength. Weird for hypertrophic obstructive cardiomyopathy, extra heart muscle should be hypeekinetic. The ra dilation (if I remember right) + the racing heart episode suggests SVT, possibly afib rvr. An electrical problem, but major stroke, heart attack risk in an aas user likely with high BP, high ldl.
The edema mentioned is concerning for an infectious issue, peri/myo carditis.
Any reduction in Ef =reduced blood through the coronary arteries, reduced O2 to the muscle makes cardiac tissue irritable and prone to electrical dysfunction like arrhythmias mentioned.
Npp/deca is notorious for lvh, add dnp induced tachycardia, maybe pericarditis from a injection infection and your SVT episode, weakened lv squeeze would track.
Your ecg may not be diagnostic but can show subtle signs of lvh (qrs amplitude v3-v6) pericarditis(low qrs amplitude, global ste, slanted tp) , hocm(dagger q) , arvdh(terminal qrs slur) , 1 AV block often precedes afib.a small infarct that killed some lv causing Hypokinesis, , bbbs, electrolyte derangement....
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