Bloodwork on Pitavastatin + Glutathione vs Without

Hey Deadpool, what are you taking during your cut. How consistent have you been with your trainging and cardio over the past 6 months.
My anabolics stayed the same, training was changed 1 week prior, I kept my volume roughly the same just split it up more evenly. to the last lab result. Otherwise all variables except for cycle support/cutting compounds/calorie deficit were the same.

My cut included T3/clen.

If you mean cycle support:
- Pitavastatin 4mg
- Eplerenone 25mg
- Telmisartan 40mg
- Jardiance 25mg
- Cialis 10mg
- Omega 3
- Astralagus
- Nac
- Tudca
- Sam-e

All these are for preventative measures, I don’t suffer from my BP, RBC, Edema
 
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Because I try to be aware of the latest, best practices for whatever I’m having addressed, I often encounter doctors reluctant to do imaging that could shed some light on whatever’s going on. Most of the time, it’s because the guidelines themselves are against imaging unless certain conditions are met.

For instance, you could have 250 LDL, 1000 CAC score, implying the widowmaker is one flight of stairs from clogging just enough to kill you, and the guidelines advise not doing an angiogram to check how blocked it is, which could reveal the need for an urgent bypass since it’s 95% blocked, unless you’re having symptoms. Never mind that the first symptom could be death.

In my mind, if there’s a 1% chance of finding a problem, I struggled to understand the rationale behind this “better not look” approach.. Ok, limiting “radiation exposure”, that makes sense. Except it applies to non or very low radiation imaging too. So that’s not the root of the guideline driven anti-imaging philosophy.

Cost to benefit ratio? Of course that’s always a factor, though when it comes to my health, if there’s any chance of a benefit from testing, I don’t give a crap about saving the insurance company money. Improve your balance sheet with some other sucker, not me. But even offering to self pay often doesn’t overcome the reluctance and insistence in following guidelines.

I finally discovered the primary reason guidelines avoid imaging unless all the factors put together meet some threshold is the harm of potentially *unnecessary procedures* resulting from seeing something in the imaging.

After giving that a lot of thought, I concluded that for a patient actively involved in their own care, it’s MUCH better to have all the information possible, then if something is discovered, make the decision as to whether it’s worth treating with some procedure or not.

There’s just no way I can see having less information is preferable to having more. At least not for a proactive, engaged patient with a couple of functioning brain cells who won’t panic, demanding surgery without weighing the risk to reward. I don’t need to be “saved from myself” by “not knowing”.

There’s no harm in seeing what’s up with your liver. It’s painless and radiation free. I’d see it as an opportunity to get a free diagnostic and be armed with more insight about your body so you can make better decisions going forward.
I truly appreciate your advice! But you might have mixed up my results with someone else (original poster?). Here are my results:


AST 75
ALT 129
Cholesterol 87
HDL 22
LDL 51
CAC score 19

Still I guess you are right that I should proceed with the ultrasound just to be informed. Thank you Ghoul!
 
I truly appreciate your advice! But you might have mixed up my results with someone else (original poster?). Here are my results:


AST 75
ALT 129
Cholesterol 87
HDL 22
LDL 51
CAC score 19

Still I guess you are right that I should proceed with the ultrasound just to be informed. Thank you Ghoul!

Wasn’t really addressing you situation specifically, just taking the opportunity to soapbox this subject since I’m going through it now :)

Trying to get a Cardiac Stress PET scan, which is cutting edge, and necessary to get a good view of my arteries. It’s the only external imaging that can see through calcified plaque to measure how open the “lumen” is along the entire length of the major vessels. I have no symptoms, but everything else in my case indicates it’s the best test for me. (If necessary I’m going to start clutching my fkng chest to give them some symptoms they can put in the record!)

About $10,000


Bracing for battle, lol.
 
These values seems amazing for someone running high doses tren/test/mast for longer periods of time.

Right @Ghoul?
First time ever testing these
 

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Yes, is this you? What’s your protocol.
This is me. Pulled bloods like you proposed.

These are my supplements.
Pita, Eplere, Jardiance, Telmisartan, B12, Safinamide, ALA, DBH are fairly recent additions.

My long term cycle support that aren’t mentioned here and were removed were: Citrus Bergamot, Tumeric/curcumin/ksm-66
 

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This is me. Pulled bloods like you proposed.

These are my supplements.
Pita, Eplere, Jardiance, Telmisartan, B12, Safinamide, ALA, DBH are fairly recent additions.

My long term cycle support that aren’t mentioned here and were removed were: Citrus Bergamot, Tumeric/curcumin/ksm-66

My rough calculations say your baseline HSCRP had to be >1.5, more likely in the high risk >3, and artery frying 5 wouldn’t be a surprise.

Any idea what it was before?

Pita 4 is doing the heavy lifting. Known to reduce HS-CRP by 50%+. Fish oil the next most helpful. Whatever the reason, thats elite low inflammation. If BP and lipids are brought into ideal range, you’ve lowered cardiovascular risk as much as is possible, and can (if you haven’t already) enter the cardiologists dream plaque regression/CV risk actually declines with increasing age zone.


Lp(a) is genetic, and below 20 the tests can be a little inaccurate, but it doesn’t matter, <20 may as well be 0 from a risk perspective.
 
My rough calculations say your baseline HSCRP had to be >1.5, more likely in the high risk >3, and artery frying 5 wouldn’t be a surprise.

Any idea what it was before?

Pita 4 is doing the heavy lifting. Known to reduce HS-CRP by 50%+. Fish oil the next most helpful. Whatever the reason, thats elite low inflammation. If BP and lipids are brought into ideal range, you’ve lowered cardiovascular risk as much as is possible, and can (if you haven’t already) enter the cardiologists dream plaque regression/CV risk actually declines with increasing age zone.


Lp(a) is genetic, and below 20 the tests can be a little inaccurate, but it doesn’t matter, <20 may as well be 0 from a risk perspective.
Sadly no baseline, however in terms of Lipids I don’t know if it matters but on my dirtiest diet and least cycle support without any statin, on 680-700mg tren, I managed a HDL of 24 and LDL of almost 0
 
I managed to pull 2 old bloodworks,
First one with good HDL was me naturally.
Second one was on 300mg test only. ( first cycle), using only Omega 3.
 

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I managed to pull 2 old bloodworks,
First one with good HDL was me naturally.
Second one was on 300mg test only. ( first cycle), using only Omega 3.
I feel like my attention to my supplementation as I continued to use gear is the main reason I managed to keep my health markers in check. Never allowed them to fall off and always obsesses on the quality and dose of my supplements.

I think I am the one of the few who only take supplements with COA’s and clinically dosed and look for the clinically accurate forms of which the supplements are derived from and also research how it’s formulated and if other ingredients/patents are required for optimal absorption.

Newest discovery is Iron heme which might fix my inherited thalassemia trait + iron deficiency which wasn’t fixed by normal enhanced iron supplements
 
I feel like my attention to my supplementation as I continued to use gear is the main reason I managed to keep my health markers in check. Never allowed them to fall off and always obsesses on the quality and dose of my supplements.

I think I am the one of the few who only take supplements with COA’s and clinically dosed and look for the clinically accurate forms of which the supplements are derived from and also research how it’s formulated and if other ingredients/patents are required for optimal absorption.

Newest discovery is Iron heme which might fix my inherited thalassemia trait + iron deficiency which wasn’t fixed by normal enhanced iron supplements

Mind blowing. I don’t know how much time you had with exposure to LDL >70, or how long since you got down to these numbers, but whatever soft plaque remains from that is, or will soon be gone, and the rest calcified into ultra stable near harmless fossils of past damage.

Since so much HS-CRP comes from arterial plaque’s “necrotic core” of trapped, yellow, rotting cholesterol goo, and yours is so low, that’s a good clue most of it’s already been vacuumed out of your arteries by HDL and disposed of.
 
On 725 test/700 tren/680 mast/10iu hgh blast. During vacation where I ate dirty, not insane surplus, around maintenance but very high saturated fats, this was my lipids on my old cycle support:

Multivitamin, Omega 3, Citrus Bergamot, KSM066, Magnesium Glycinate, Tumeric/Curcumin, Sam-e, Astragalus, red rice yeast + comq10, Cialis 10mg, dandelion root 2g, Tudca, NAC, 2g metformin ir, 2g metformin XR, 30IUs Lantus, gda clinically dosed. (split to 2 injections to avoid ir and lower blood glucose levels).

However clearly I dropped the excessive metformin and realized lantus was useless in my case.

Btw: I naturally have high liver enzymes, this was post anavar use too. However this thread is about my liver enzymes so y’all clearly know how I am approaching it rn.
 

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On 725 test/700 tren/680 mast/10iu hgh blast. During vacation where I ate dirty, not insane surplus, around maintenance but very high saturated fats, this was my lipids on my old cycle support:

Multivitamin, Omega 3, Citrus Bergamot, KSM066, Magnesium Glycinate, Tumeric/Curcumin, Sam-e, Astragalus, red rice yeast + comq10, Cialis 10mg, dandelion root 2g, Tudca, NAC, 2g metformin ir, 2g metformin XR, 30IUs Lantus, gda clinically dosed. (split to 2 injections to avoid ir and lower blood glucose levels).

However clearly I dropped the excessive metformin and realized lantus was useless in my case.

Btw: I naturally have high liver enzymes, this was post anavar use too. However this thread is about my liver enzymes so y’all clearly know how I am approaching it rn.

Red Yeast Rice is hepatoxic (many confirmed cases of liver damage) and it’s literally a statin, but the least effective, worst side effect causing type. You’ll never want that in your stack again.
 
Thanks for all the analysis and suggestions @Ghoul. Appreciate it alot <3

I’m just tweaking around the edges, you were already doing really well. Don’t let anyone ever make you feel like it’s “extreme”. These levels, and striving for regression is where all the data from mainstream, top of the field research organizations is pointing too, and will be part of the guidelines years or a decade from now. You’re just getting the benefits in early, when they’ll provide the greatest benefits.

Consider getting a calcium score. If it’s a positive number, that suggests some degree of ischemia (narrowing of the lumen, aka the “pipe opening”. It often has to be 90% or more narrowed for symptoms to develop.) It won’t worsen with your lipid numbers, there’s no more plaque accumulating, but it’s a residual risk (something could clog a calcium narrowed lumen (god forbid in the widowmaker for instance) more easily than a fully opened lumen. This tends to happen in old age more, but some infections and other illnesses can put you at risk for a piece of endothelium to slough off and create a blockage. Thats why risk is a heart attack or stroke is never zero.

The real benefit of knowing this is that you can focus on pushing your heart to the absolute max safe limits a few times a week.

This creates “ischemic stress”, where the heart doesn’t get enough oxygen. Sounds scary but it isn’t, just counterintuitive, but safe as long as you don’t get any symptoms.

This stress triggers a process called “ischemic adaptation”, where the arteries feeding the heart generate new blood vessels that bypass the narrowed areas. This increases exercise endurance, but more importantly if there’s ever a sudden blockage, having these “alternate route” vessels greatly increases your chance of survival and escaping heart / brain damage. Obviously do your own research so you’ll know how to do this safely, but even after addressing 95% of the risk factors like you have, it’s good to know of and go after the remaining “residual risk”. The younger you do the ischemic adaptation exercises, the greater capacity for new vessel growth you have.
 
Sounds nuts so here’s the best illustration I can find at the moment of the a,b,c,d bypass vessel growth process. For some reason they start with small bypass vessels already in place (new ones sprout from the same stimuli), but it illustrates how increasing stress stimulates them to grow into large blood flow capacity arteries:

IMG_3474.webp
 
Noted, def sounds interesting and worth checking out.

I try to optimize everything I can to be able to run as much gear as I can safely.

I experience 0 sides outside of water retention, I naturally like to retain some water in my face and lower abdomen, otherwise I feel 0 difference on and off of anabolics.

So being able to run high doses and also have perfect bloodwork is amazing. I just run the same 4 compounds (tren/test/mast/gh) and maximize everything possible pathway I can and I don’t need to mess with anything else regardless of being in a bulk/cut. Which makes it very easygoing to plan.

I have to resist the urge to go 1g of each compound for fun.
 
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