Phil Heath's abdominal surgery

Who is this .... commenting about PHs "health issue" and why would a physician "come to me (him)" to explain the "seriousness of Phils condition"?

People live with hernias for decades and often do fine but request surgery
to correct the protrusion for cosmetic
reasons and a "three inch hernia" is no exception.

Finally hernias have a recurrence rate
ranging bt 0-100% depending upon a variety of factors and umbilical hernias are at the higher end .

Jim
I dont think it was about the hernias, it was about the "gut" phenomenon.
 
Ill throw my observations into the mix and they parallel Wunderpus's observations.

When i add insulin my abdominal area expands. When i stop taking insulin it shrinks back down. Insulin seems to be the major factor. Solo GH has no impact on my gut that ive noticed. Add in slin though and things seem to thicken up in my abdomen, but its only temporary.
 
Oh god, for christ sakes it's HGH and insulin abuse. Period. I love all of the excuses ppl have now....."the massive amounts of food" hahaha.
 
What're your thoughts on the cause of this "distended" or "GH gut"? Do you believe it's from GH, IGF-1, AAS, overeating, insulin or some other cause?

Although tempting considering the poly-pharmacy practices of Professional BB its impossible to isolate drug specific adverse effect relationships IME

That being said its no coincidence "the gut" appeared and the V-taper of Arnies era disappeared as GH became more readily available and the emphasis became one of "mass at all costs".

This observation is further supported by autopsies in those with Giganticism, in which organomegaly is a commonplace, if not expected, finding. Albeit of a lesser magnitude I suspect a "thickened core" may contribute to this appearance as @mands opined.

Finally bc the side effects of AAS are amplified considerably when combined with GH, the danger is all the more real.

To that end what we "see" on the outside (as in the "gut") is likely to be the tip of the iceberg and the deaths of late are likely to be a precursor, (or warning) of whats to come IMO.

JIM
 
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Dorian Yates, all the proof you need as to when it began. Although not as pronounced it was still there
He talks about it. Says that the issues only arose after the introduction of insulin. Now, it could be just a coincidence (i.e. the growth was already occurring when he added the insulin), or the insulin fueled it faster. Nonetheless, you can see it towards the end of Dorian's career.
 
He talks about it. Says that the issues only arose after the introduction of insulin. Now, it could be just a coincidence (i.e. the growth was already occurring when he added the insulin), or the insulin fueled it faster. Nonetheless, you can see it towards the end of Dorian's career.

Its not a coincidence ffs... I know that you like cold hard scientific supported evidence, but lets be real here... Its the GH, Slin and the peptides....
 
A

Finally bc the side effects of AAS are amplified considerably when combined with GH, the danger is all the more real.

To that end what we "see" on the outside (as in the "gut") is likely to be the tip of the iceberg and the deaths of late are likely to be a precursor, (or warning) of whats to come IMO.

JIM

In closing, this article was published in this months issue of Muscular Development.

Regs
JIM
 

Attachments

Oh god, for christ sakes it's HGH and insulin abuse. Period. I love all of the excuses ppl have now....."the massive amounts of food" hahaha.
Well i think of it like this...whats phil heath go for? 260 on stage? 300 offseason? At what? 5'freakin9? What happens if you take another 5'9 dude at 260-300lbs? Hes going to be fat. His stomach is going to be extended. Presumably from the food. I think thats a safe assumption right? And oddly the only one with actual merit. As in food causes stomach distention. What happens if that same 5'9 fat dude goes on a diet? Holy bologni! His stomach gets flatter!!! Presumably because hes eating less food, right?
 
Well i think of it like this...whats phil heath go for? 260 on stage? 300 offseason? At what? 5'freakin9? What happens if you take another 5'9 dude at 260-300lbs? Hes going to be fat. His stomach is going to be extended. Presumably from the food. I think thats a safe assumption right? And oddly the only one with actual merit. As in food causes stomach distention. What happens if that same 5'9 fat dude goes on a diet? Holy bologni! His stomach gets flatter!!! Presumably because hes eating less food, right?
I believe the HGH and insulin changes the metabolic rate to a point where physiologically something has to give to be able to consume that many calories without straight up fat gain. Essentially these guys are still leaner than usual at a heavier weight. I agree with you to a point, but with that being said I've seen middleweight and light heavy competitors with terrible guts also.
 
While food or intestinal gas (the latter being the primary cause of post-prandial abdominal distention) may account for some of the abdominal distention seen in Pros, since many of these "belly PICs" are obtained during an active competition, the impact of food would be minimal as it's unlikely a Pro would gorge themselves during this interval, for the very reasons you described.

(As an aside therein lies the difficulty of using a terms such as "bloat, blast, cruise etc" to define a drugs adverse effect that is NOT described in the medical literature, it has different meanings for different folk).

Consequently, IF the process I referred to earlier is/was the result of organomegaly the condition would be chronic and remain relatively constant upon awakening with an empty stomach.

Another comparison is what Arnie coined as the "Vacumn pose", something few if any modern day BB are capable of ------ the most likely evidence based reason remains Organomegaly IMO

Jim
 
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He talks about it. Says that the issues only arose after the introduction of insulin. Now, it could be just a coincidence (i.e. the growth was already occurring when he added the insulin), or the insulin fueled it faster. Nonetheless, you can see it towards the end of Dorian's career.

Based upon diabetic research I'm unaware of ANY correlation or association between the use of Slin and "gut distention".

In fact the primary etiology becomes even more apparent as we contrast the disproportionate changes in TBF and LBM in those using GH on a chronic basis, since Slin especially in high doses, is associated with enhanced visceral adiposicity, which is the antithesis of what is noted with GH.

The point, the evidence strongly suggests GH/IGF as the final common pathway, and whether Slin exacerbates this condition in ""normal" folk using GH (assuming both are used in supraphysiologic dosages) remains unclear.

However bc the effects of IGF and Slin are complimentary physiologically in many respects, I doubt this is the exception.

Stated another way, while Slin may be the horse GH/IGF is the carriage.

Regs
Jim
 
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Based upon diabetic research I'm unaware of ANY correlation or association between the use of Slin and "gut distention".

In fact the primary etiology becomes even more apparent as we contrast the disproportionate changes in TBF and LBM in those using GH on a chronic basis, since Slin especially in high doses, is associated with enhanced visceral adiposicity, which is the antithesis of what is noted with GH.

The point, the evidence strongly suggests GH/IGF as the final common pathway, and whether Slin exacerbates this condition in ""normal" folk using GH (assuming both are used in supraphysiologic dosages) remains unclear.

However bc the effects of IGF and Slin are complimentary physiologically in many respects, I doubt this is the exception.

Stated another way, while Slin may be the horse GH/IGF is the carriage.

Regs
Jim
This is a great way to put what I always theorized
 
I really can't stand when a bodybuilding commentator for.tje Olympia or Arnold says,"He needs to control that midsection." That shit goes all over me man. Control the midsection? Gtfo. The guy should get last place, not 1st
 

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