Based upon diabetic research I'm unaware of ANY correlation or association between the use of Slin and "gut distention".
In fact the primary etiology becomes even more apparent as we contrast the disproportionate changes in TBF and LBM in those using GH on a chronic basis, since Slin especially in high doses, is associated with enhanced visceral adiposicity, which is the antithesis of what is noted with GH.
The point, the evidence strongly suggests GH/IGF as the final common pathway, and whether Slin exacerbates this condition in ""normal" folk using GH (assuming both are used in supraphysiologic dosages) remains unclear.
However bc the effects of IGF and Slin are complimentary physiologically in many respects, I doubt this is the exception.
Stated another way, while Slin may be the horse GH/IGF is the carriage.
Regs
Jim