ChemBB
Member
This is only partially correct80mg trest D (amino asylum?) is about 50mg of actual trest, and should be similar to estradiol from 140mg/wk of Test C which would put most people in a perfect upper normal range in the absence of any AI. Suspect your libido is good and joints feel good despite what is seemingly a crashed e2.
EQ has more A-ring similarities to exemestane/amatestane/formestane than other AAS, but is still more likely to work as a competitive AI and/or source of estrone that effectively acts as a (non-selective) SERM by binding to ER and preventing the much more active estradiol from binding.
You could try getting a standard/cheap ECLIA estradiol test, I think it will detect the 7α-me-e2 but will also mis-detect trenbolone as estradiol. That was the old school way of confirming that tren was real.
(I need to write a dedicated thread on Trest and aromatization)
This is spot-on about EQ. It has both a lower binding affinity (Km) and slower and rate of conversion (Vmax) at Aromatase than Testosterone. If you want to ballpark it, let's call it 20-30% of Test.
Boldenone Metabolites: 5a-reductase -> DHB (Dihydroboldenone) Aromatase -> Estrone (E1) (Weak estrogen, limited conversion to E2 via 17b-HSD) 3b-HSD -> 1-Androstenedione (DHB precursor, via 17b-HSD) 17b-HSD -> 1-Androstenediol (Weak androgen. DHB + 1-Androstenediol convert into each other cyclically) 16a-hydroxylase -> 16a-Hydroxy-boldenone (E1 analog, weak estrogen)But the bit about Trest's estrogenic potency is not correct. I have to head to the gym but I can write a proper response with cited sources when I get back

