Pita / Zetia / Vascepa vs test / tren / mast / var (bloodwork)

[RockyP]

Will get straight to it. As promised the battle royale between the lipid destroying DHT’s and Tren and pita / Zetia.

Sustanon 150 eod
Mast P 140 eod
Tren ace 60 eod
Anavar 40-50 daily (for last 10 days prior to labs)

Cardiac regimen
Pita 4 mg
Zetia 10 mg
Vascepa 4 g
Nattokinase 4000 FU
Reta 5 mg (not cardiac but plays a role in metabolism)

LDL 33
Hgb 16
HCT 48
triglycerides 57
HDL 11 (thanks anavar)
Lp(a) < 9
ApoB 53
Sensitive E2 was 43 with no AI

All other parameters normal including CRP, GGT, AST, ALT, and cystatin C.

Other support Supps are the usual berberine, astragalus, ALA, K2 D3, ubiquinol CoQ10, and I’m prob forgetting some.

Will be running these labs again off of the tren and anavar. Want to see where the HDL sits on just test / mast. But with an LDL of 33, it’s not such an issue. Today is the last day of anavar and I’ve got maybe 3 days left on tren.

Thank you to @Ghoul for sharing all the cardiac information.

Also, all of those cardiac parameters IMPROVED (not the HDL, that was 23 on test / primo) since the last draw WHILE adding Tren, mast, and var.

 

[Ghoul]

Will get straight to it. As promised the battle royale between the lipid destroying DHT’s and Tren and pita / Zetia.

Sustanon 150 eod
Mast P 140 eod
Tren ace 60 eod
Anavar 40-50 daily (for last 10 days prior to labs)

Cardiac regimen
Pita 4 mg
Zetia 10 mg
Vascepa 4 g
Nattokinase 4000 FU
Reta 5 mg (not cardiac but plays a role in metabolism)

LDL 33
Hgb 16
HCT 48
triglycerides 57
HDL 11 (thanks anavar)
Lp(a) < 9
ApoB 53
Sensitive E2 was 43 with no AI

All other parameters normal including CRP, GGT, AST, ALT, and cystatin C.

Other support Supps are the usual berberine, astragalus, ALA, K2 D3, ubiquinol CoQ10, and I’m prob forgetting some.

Will be running these labs again off of the tren and anavar. Want to see where the HDL sits on just test / mast. But with an LDL of 33, it’s not such an issue. Today is the last day of anavar and I’ve got maybe 3 days left on tren.

Thank you to @Ghoul for sharing all the cardiac information.

Also, all of those cardiac parameters IMPROVED (not the HDL, that was 23 on test / primo) since the last draw WHILE adding Tren, mast, and var.

So you’ve essentially turned off the flow of incoming plaque, the most important factor.

Obviously HDL-C is well below guideline “healthy” levels. So this would suggest that the main problem now is plaque regression that might otherwise be happening is impaired. After all, it’s HDL that “reverse transports” cholesterol out of the arteries for disposal by the liver.

But HDL-C doesn’t really tell us much when LDL-C is so low.

That’s because it’s a measurement of the cholesterol “load” within HDL, not how many HDL “dump trucks” you have or how well they’re functioning. You might just have a low amount of arterial plaque available to be reversed, so not much cholesterol for HDL to remove, resulting in a low HDL-C.

If you want to measure how well your plaque removal mechanism is functioning HDL-C isn’t the marker.

Checking HDL-P (particle count) will tell you how many HDL “trucks” are available to carry cholesterol out of your arteries, and APO-I will tell you whether they’re working well or broken down.

Not absolutely necessary, but you might be pleasantly surprised to find HDL-P is high and APO-I is adequate, and plaque is regressing, or not. More out of curiosity than anything else, always nice to know what’s going on.

 

[RockyP]

So you’ve essentially turned off the flow of incoming plaque, the most important factor.

Obviously HDL-C is well below guideline “healthy” levels. So this would suggest that the main problem now is plaque regression that might otherwise be happening is impaired. After all, it’s HDL that “reverse transports” cholesterol out of the arteries for disposal by the liver.

But HDL-C doesn’t really tell us much when LDL-C is so low.

That’s because it’s a measurement of the cholesterol “load” within HDL, not how many HDL “dump trucks” you have or how well they’re functioning. You might just have a low amount of arterial plaque available to be reversed, so not much cholesterol for HDL to remove, resulting in a low HDL-C.

If you’re you want to measure how well your plaque removal mechanism is functioning HDL-C isn’t the marker.

Checking HDL-P (particle count) will tell you how many HDL “trucks” are available to carry cholesterol out of your arteries, and APO-I will tell you whether they’re working well or broken down.

Not absolutely necessary, but you might be pleasantly surprised to find HDL-P is high and APO-I is adequate, and plaque is regressing, or not. Still, always nice to know what’s going on.

Thank you sir. I’ve always had low HDL even on TRT. I do want to see where I sit on test / mast and then just test cruise. But I’m very pleased with these LDL, ApoB, and Lp(a) numbers. And this is without nexletol. I still have it on deck if needed. This is about as harsh a cycle as I’ll ever run.

 

[Ghoul]

Thank you sir. I’ve always had low HDL even on TRT. I do want to see where I sit on test / mast and then just test cruise. But I’m very pleased with these LDL, ApoB, and Lp(a) numbers. And this is without nexletol. I still have it on deck if needed. This is about as harsh a cycle as I’ll ever run.

Despite driving “LDL lower regardless of how” is better, the one caveat with Bemp long term is that tendon risk. It’s very low, and only seen in older ages so far, but it’s definately there. With my LDL @ 24 it would only get me a few points lower so not worth even a tiny added risk for unmeasurable gain.

But if I wasn’t using a statin for whatever reason, or didn’t have Repatha in my stack, Bemp could lower LDL much more and tip the balance in its favor. I just have nothing to gain from it with current levels.

 

[RockyP]

Despite driving “LDL lower regardless of how” is better, the one caveat with Bemp long term is that tendon risk. It’s very low, and only seen in older ages so far, but it’s definately there. With LDL it would only get me a few points lower so not worth even a tiny added risk for unmeasurable gain.

But if I wasn’t using a statin for whatever reason, or didn’t have Repatha in my stack, Bemp could lower LDL much more and tip the balance in its favor. I just have nothing to gain from it with current levels.

That’s how I feel also, especially given these labs on a stack known to cause harsh dyslipidemia. I purposely tested labs on mast / tren / anavar to see how the pita and zetia would handle it and I’m impressed.

 

[Ghoul]

That’s how I feel also, especially given these labs on a stack known to cause harsh dyslipidemia. I purposely tested labs on mast / tren / anavar to see how the pita and zetia would handle it and I’m impressed.

For anyone reading this I want to be clear that I’m not advising against Bemp, it has a very good safety record with no sides ever developing for most people even after decades of use.

It’s just that once LDL is in the 30s, Bemp won’t bring it down very much (or at all). So I’d reserve it as the last add on if you’re >40 after using everything else that will be in your lipid stack.