I have been thinking of a similar approach. What ratio of TNE to ester are you using? Also, is it okay to mix a test ester with TNE in the same syringe?
I was using 15mg Test E (0.05cc @ 300mg/ml) + 5mg TNE (0.1cc @ 50mg/ml). I am a night person, so was shifting the peak times ie TNE shot into the afternoon so it peaked during my workout into the evening, then would fade off by the time I was ready for bed.
Basically a 3:1 mg ratio of esterified test (specifically cypionate or enanthate, which is what most use) to TNE should give the 30% diurnal release variation iseen with young men.
I since changed up my regimen and increased my MENT dose to 6mg, and am currently using just a tiny bit of test prop (7mg/day) as part of a RIP (test prop + mast prop + tren ace) mix, but will still add TNE for preworkout effect. If I feel my DHT or estrogen is borderline low I take 12.5-25mg, other days just 5mg. I have needed only minimal to no aromasin - last two weeks I took only 6.25mg and I wasn't sure I needed it.
6mg/day MENT doesn't sound like a lot, but a replacement dose (for estradiol) is about 0.5mg/kg per week, so I'm basically getting enough estrogen from that alone. Probably more, as I tend to be a high converter despite being lean - maybe because I was a chubby teenager and/or am old idk. So any estrogen from test is just extra - taking the test only to have a bit of DHT as I do feel slightly better with it than without; DHT derivatives and even oral or injectable DHT do not fulfill whatever needs there are for DHT made as a test metabolite, because they are not locally produced in a tissue specific manner.
Btw this is why MENT 'failed' in the replacement studies. Doses were set by ability to maintain lean body's weight and libido, at < 2 mg/day, with reference being 200mg/wk test C ie a generous TRT dose. The 7a-methylestradiol is a true biologic replacement for estradiol, it has 102% affinity for ERa/ERb, is locally produced by aromatase, and seems to be more effective than estradiol at transactivation - at least in a T47dco breast cancer cells - an estrogen resistant/defective cell line so maybe not be best way to predict transactivation in healthy human cells. But from anecdotal it is clearly some multiple. of estradiol, plus it is all free as the 7a methyl group blocks any SHBG binding - that's more important with androgens, but SHBG does bind a little estradiol too.
At the studied doses there was some bone loss observed particularly at the lower dose ranges - indicative of insufficient estrogen. If they had added two more MENT dose ranges ie 3200 & 6400mcg per week, estrogen would have been sufficient to maintain bone mass, AND the participants would have gotten jacked - similar to 500-750mg/wk testosterone in anabolism, which is a first or second cycle typical for the type of people here. So of course, physique enhancement is not an allowable endpoint in medical research, so it was deemed verboten and discarded, until UGLs began making it.
As an independent thinker I saw the potential here - basically HRT with about 10x the anabolism with much less stimulation of prostate and hair loss than test. I was hoping to feel perfectly fine without any test, but for me that was not the case. But just 50-70mg per week and 3x250-300iu of HCG is enough for adequate DHT and upstream adrenal hormone precursors.