malfeasance
Member
The personal ceiling changes with age, too. It goes down, down, down . . .when you can no longer safely and sustainably increase your dose you have reached your personal ceiling.
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The personal ceiling changes with age, too. It goes down, down, down . . .when you can no longer safely and sustainably increase your dose you have reached your personal ceiling.
Why nandrolone too? For the same neurotoxicity?
Because it's essentially probably anti GABAergic and probably lowers dopamine neurotransmission in some key brain areas, and this can persist even after cessation. If you've ever seen somebody unable to stand still, being "shifty" (for lack of a better word) and anxious while on nandrolone, these are probably the reasons why. It also has the potential to cause HPA axis hypofunction.
It's basically a depressant and anxiogenic with a higher neurotoxicity load then other aas. This is what distinguishes it from other aas, but all aas are bad for your brain (in supra-physiological dosages) mind you ...
The only possible consolation for nandrolone users is that most of the statements mentioned in the first paragraph are based on studies conducted in rats. So, if someone wishes to be in denial, that might offer a limited basis for doubt.
No experience with Deca/Nand but my understanding is that older folks use it for its joint/tendon anti-inflammatory properties? That seems like a valid reason if you have said issues no?
Deca is also supposed to be easy on the HDL/LDL right? This is of interest to me because Test-C did do a number on my HDL/LDL at 500mg. So im trying to figure out if I can replace some of the Test-C with something else.
I assume dosages for us older folks are rather tame. Maybe around 100mg a week? Whats your take on that? Still not worth it given the neurotoxicity?
Imo, no, not at any dose no, but more so for it's general negative neuromodulatory properties outlined in my first paragraph then it's neurotoxicity at the dosages you mentioned. Aas are pretty shit drugs, I hope they get replaced by something better sooner then later.
Imo, no, not at any dose no, but more so for it's general negative neuromodulatory properties outlined in my first paragraph then it's neurotoxicity at the dosages you mentioned. Aas are pretty shit drugs, I hope they get replaced by something better sooner then later.
Thanks. I was almost convinced Deca was a good option but I clearly need to do more research.
