HGH cruise dose

duddy707

New Member
Do you guys drop your GH dose when you go into a cruise? I’m on 5-6iu daily for the last few months, is there any reason to drop that to 2-3iu when I lower my gear dosages? Tysm!
 
I started gh about four years ago , getting ready for a competition, of course. I was forty years

Since then I’ve used 1,and 1.5 ius year round, but for the first time with China restrictions, very popular gh company said they were out, and let it go

Most I’ve done in prep was 3 ius

In competition prep and over eight weeks out and still haven’t gotten any. I’ve heard the crogmagn facial features begin at 5-6 ius but I don’t know , Dorian and Jays face may look a bit overgrown. Ronnie’s not so much. It could also depend on your race, some skull shapes and gh may interact differently, again idk
 
introduced recently so i havent had a chance to cruise on it yet but plan is to blast 4iu and cruise 2iu :)
 
Is that a big issue when running 4ius year round?

The way risk is measured is by IGF-1 level for your age, not the dose of rHGH.

Find your ULN (upper limit of normal) IGF based on assay type(ie lab that performed the test):

IMG_2380.webp

Your risk of developing signs of acromegaly depends on how much higher than the ULN you are:

IMG_2381.webp

At 1.3x ULN acromegaly features could begin to develop at between 2-5 years if you sustain IGF this high.

At 1.5x initial features can develop at 2 years.

2x starting at 1 year.

Above 2x acromegaly can begin to develop in less than a year,

First is soft and connective tissue, lips, nose, ears, organs, fully (lips) or partially (cartilage) reversible if you stop in time.

Then bone changes slowly over time, forehead, chin, teeth begin to misalign, irreversible.

It happens so slowly even family members often miss it for 5+ years.
 
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The way risk is measured is by IGF-1 level for your age, not the dose of rHGH.

Find your ULN (upper limit of normal) IGF based on assay type(ie lab that performed the test):

View attachment 345268

Your risk of developing signs of acromegaly depends on how much higher than the ULN you are:

View attachment 345269

At 1.3x ULN acromegaly features could begin to develop at between 2-5 years if you sustain IGF this high.

At 1.5x initial features can develop at 2 years.

At 2x at 1 year.

Above 2x acromegaly can develop in less than a year,
great explanation, thanks for the info. Imma test it in a week or 2, I think at 4ius, realistically I'd be around 1.3 - 1.8 at my age (21) but we'll have to see.
I'm using lr3 40mcg as well for 6weeks so it'll be interesting to see the results
 
great explanation, thanks for the info. Imma test it in a week or 2, I think at 4ius, realistically I'd be around 1.3 - 1.8 at my age (21) but we'll have to see.
I'm using lr3 40mcg as well for 6weeks so it'll be interesting to see the results

You can see on the chart your levels can be much higher, and even when exceeding them, excess IGF can be "absorbed" into normal growth so problems would take much longer to develop at 20 than someone at 50 running 1.5x.

With age the excess IGF becomes increasingly likely to cause abnormal circus freak growth.
 
You can see on the chart your levels can be much higher, and even when exceeding them, excess IGF can be "absorbed" into normal growth so problems would take much longer to develop at 20 than someone at 50 running 1.5x.

With age the excess IGF becomes increasingly likely to cause abnormal circus freak growth.
yep, outside of LVH, organomegaly, increased cancer.
I always wondered if the main precursor to those bodybuilder's bubble guts were the insane doses of GH or was it also the insulin. I feel like GH would softly contribute to it but insulin would be the final nail in the coffin as you need to eat a shit ton of calories and bloating and igf1 ramp up would likely be the main cause, I think.
Cuz honestly that bubble gut is fucking hideous IMO.
 
yep, outside of LVH, organomegaly, increased cancer.
I always wondered if the main precursor to those bodybuilder's bubble guts were the insane doses of GH or was it also the insulin. I feel like GH would softly contribute to it but insulin would be the final nail in the coffin as you need to eat a shit ton of calories and bloating and igf1 ramp up would likely be the main cause, I think.
Cuz honestly that bubble gut is fucking hideous IMO.

Insulin probably plays a role (use a glp with hgh if you can). monitor glucose, no need to repeat the mistakes of the past you can see living examples of.

Also though, intestines grow fast with rHGH, first thickening, then lengthening.

These can both be positive. Thickening enhances the barrier that prevents endotoxin from leaking from intestines into your bloodstream, reducing systemic inflammation. Lengthening enhances nutrient absorption. Too much intestine overgrowth (they won't ever stop growing with IGF too high above ULN) and you get Palumboism.

Just remember. if it does happen, pink bikini shorts can pull the look together so no one will notice.

IMG_2382.webp
 
The way risk is measured is by IGF-1 level for your age, not the dose of rHGH.

Find your ULN (upper limit of normal) IGF based on assay type(ie lab that performed the test):

View attachment 345268

Your risk of developing signs of acromegaly depends on how much higher than the ULN you are:

View attachment 345269

At 1.3x ULN acromegaly features could begin to develop at between 2-5 years if you sustain IGF this high.

At 1.5x initial features can develop at 2 years.

2x starting at 1 year.

Above 2x acromegaly can begin to develop in less than a year,

First is soft and connective tissue, lips, nose, ears, organs, fully (lips) or partially (cartilage) reversible if you stop in time.

Then bone changes slowly over time, forehead, chin, teeth begin to misalign, irreversible.

It happens so slowly even family members often miss it for 5+ years.
Thanks for taking your time and posting this. This is exactly what I was looking for.
Been hesitant to start , but this a good baseline .
 
Thanks for taking your time and posting this. This is exactly what I was looking for.
Been hesitant to start , but this a good baseline .

I think it's a become relevant question for a lot of us because until fairly recently, the idea of running rHGH above therapeutic doses (<2 iu) continuously for years, or indefinitely, was limited to a tiny minority given the cost. And we've seen some of the potential effects on those who have gone down that road.

It would be nice to know specifically how much and how long they used it, but few are even willing to acknowledge they did it at all (ahem, Stallone).

So the best we can do is extrapolate from the IGF levels doctors use to safely guide long term rHGH treatment, which is for life, and also the levels endocrinologists start to suspect acromegaly from tumors and when they feel they have to do something to bring them down via surgery or whatever. We also have a little evidence of elevated IGF levels, and how long they were at that level before symptoms started to appear.

It's all a sophisticated "best guess", and hopefully I've erred on the side of caution (ie, under 1.2x ULN it doesn't seem anyone develops axromegalic features)

It may be wrong, way over cautious or (god forbid) too much, but until there's more data on what the establishment would call "rHGH abuse" it's better than just dosing by feels and hoping for the best.

ESPECIALLY as the most well known bro-science advice on dose has been "As much as you can tolerate and afford".
 
The way risk is measured is by IGF-1 level for your age, not the dose of rHGH.

Find your ULN (upper limit of normal) IGF based on assay type(ie lab that performed the test):

View attachment 345268

Your risk of developing signs of acromegaly depends on how much higher than the ULN you are:

View attachment 345269

At 1.3x ULN acromegaly features could begin to develop at between 2-5 years if you sustain IGF this high.

At 1.5x initial features can develop at 2 years.

2x starting at 1 year.

Above 2x acromegaly can begin to develop in less than a year,

First is soft and connective tissue, lips, nose, ears, organs, fully (lips) or partially (cartilage) reversible if you stop in time.

Then bone changes slowly over time, forehead, chin, teeth begin to misalign, irreversible.

It happens so slowly even family members often miss it for 5+ years.
If one were to only run higher HGH for a shorter period of 6mos or less, then drop to a dose that is <1.2 ULN, does that mitigate the risk? Or is there a permanent impact from the higher dose, even if not visible and if you ever resume at a higher dose, you are essentially picking up where you left off in terms of potential side effects?
 
If one were to only run higher HGH for a shorter period of 6mos or less, then drop to a dose that is <1.2 ULN, does that mitigate the risk? Or is there a permanent impact from the higher dose, even if not visible and if you ever resume at a higher dose, you are essentially picking up where you left off in terms of potential side effects?
come on man , this has been exhaustively discussed and if you actually used the search function you're gonna find the answer, but im sure ol Ghoul will chime in.
 
The way risk is measured is by IGF-1 level for your age, not the dose of rHGH.

Find your ULN (upper limit of normal) IGF based on assay type(ie lab that performed the test):

View attachment 345268

Your risk of developing signs of acromegaly depends on how much higher than the ULN you are:

View attachment 345269

At 1.3x ULN acromegaly features could begin to develop at between 2-5 years if you sustain IGF this high.

At 1.5x initial features can develop at 2 years.

2x starting at 1 year.

Above 2x acromegaly can begin to develop in less than a year,

First is soft and connective tissue, lips, nose, ears, organs, fully (lips) or partially (cartilage) reversible if you stop in time.

Then bone changes slowly over time, forehead, chin, teeth begin to misalign, irreversible.

It happens so slowly even family members often miss it for 5+ years.
Why is it that the IGF level necessary to cause those issues varies with age? You’d think if 400 was safe from those side effects at age 22, it would be safe also at age 50.
 
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