For me yes. Worse lipids on a lower dose of primo than a higher dose of tren.
MESO-Rx
Anabolic Steroids
Had a Heart Attack today at age ~30. Be safe guys.
- Thread starter ChemBB
- Start date
Kind of scary that your lipids lol that good but you just had a heart attack.
dinfar1337
New Member
its very easy to make those models with simple tools in word. we play with it from elementary school here in eu.
3d and adding lines / shapes and sizing as you want.
you could also just steal some "3d sketch drawing round pill" or whatever shape you want and use Remove Background from Image for Free – remove.bg
dinfar1337
New Member
something like this could look nice
ChemBB
Member
I stopped using AI about 5 years ago and will never use them again
Ah, that's from hospital mate!
I don't think so, but maybe I'm mistaken?
They did a 1968 study giving female breast cancer patients 1,200mg Primo / wk and that wasn't one of the acute sides mentioned
What does your estrogen hover around when. You did 350 for that period
ChemBB
Member
At 300-350 Test by itself, E2 = ~40-90 generally
When I did 250 Test + 200 Mast E for 8 months back in 2017, my E2 was 19
No sides with it getting up to 90?
Also how was your HDL on the 19 bloofwork? When I run test and primo (even a baby dose of prim) it lowers my estrogen so much it ruins my lipids.
ChemBB
Member
No sides as far as I can tell -- I have gyno from when I was 17 and first started gear. (I kept upping Dbol dose until I was taking 100mg a day because I wasn't sure if it was "doing anything". </facepalm>)
So if my E2 gets too high, I can feel my nips burning + gyno growing, which I don't.
I have libido issues but it's been that way forever regardless of what I take. Pretty sure it's from years of drug addiction messing up my neurochemistry.
I wish I could tell you. I didn't check my lipids EVER when I was young, only started in the last few years =(
I am sorry for you but you didn't write anything about your lifestyle.
Maybe you run with high BP since a long time, maybe you eat tons of saturated fats.
Stay safe doesn't mean anything.
Banana Joe
Member
Were you then sure it "does something" when you had hit 100mg?
Those were the golden years for anabolics. They used them to treat anything. No matter what illness you had, doctors gave you a chance to get swole.
I wish that had never changed. Just imagine what kind of steroids we would have by now, had they poured GLP1-drug kind of money into researching them.
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ChemBB
Member
Yeah, because I got gyno almost instantly LMAO
Went from no sides, not sure if it was fake sugar powder, to suddenly I wake up and my nips burning, hurting and itching
That's when I realized it was definitely dbol.
I didn't want to be sad this early in the morning.... =(
OldGHGuy
Member
Received my bloods back this week and have some relavant results attached. Running 250 Test, 150 Primo, 2.4 GH, 5 mg Tirz. A few interesting points including improvements from previous when running just 140 Test TRT. 40mg Atorvastatin year round.
I did find out this is too much Primo for this level of Test for me as my sensitive E2 came back at 16. Strangely no joint issues but stomach bloating that I believe I wrongly attributed to a virus but now believe it was the low E2
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Banana Joe
Member
And there was me, when I was even a few years older, keeping my 2x5mg Dbol run to 5 weeks, because I was worried about my liver. When the general forum consensus was that 8 weeks are a safe length for such a cycle, obviously at higher doses than mine.
I haven't given up that there will not be an anabolic renaissance.
Just imagine cheap gene testing and AI designing afforable custom anabolics for every man.
PS: Thanks again for putting this all out there! There will be that one guy, that you will never know about, that you scared into being more more diligent with his health.
Ghoul
Member
Not bad man!
If you switch to Pitavastatin, you may see a 10+ point boost to HDL, a very clinically significant increase that would make plaque regression move at a good pace with your fairly low LDL. I saw an 11 point jump from 39 to 51 on 200mg Test. Unless you're on Atorvastatin due to kidney disease, Pita is superior.
If anyone needs inspiration, my lipids from September 2024:
To September 2025:
This is with Pitavastatin 4, Ezetimebe, and Repatha. The Repatha is only adding 10-12% more improvement to the other two though.
After these, and stress echo + CPET results were sent, my preventative cardiologist added a note:
"You have undergone a complete cardiometabolic risk inversion. From a 10-year ASCVD risk near 18%, you now present with optimal blood pressure, an LDL profile in the lowest 0.1% of the population, and endurance-level aerobic fitness. Your current risk is...
To September 2025:
This is with Pitavastatin 4, Ezetimebe, and Repatha. The Repatha is only adding 10-12% more improvement to the other two though.
After these, and stress echo + CPET results were sent, my preventative cardiologist added a note:
"You have undergone a complete cardiometabolic risk inversion. From a 10-year ASCVD risk near 18%, you now present with optimal blood pressure, an LDL profile in the lowest 0.1% of the population, and endurance-level aerobic fitness. Your current risk is...
sionnach
Banned
I'm sure you've mentioned this in one of your many posts, but what HDL-promoting supps do you take personally (fish/krill oil, omega 3, dietary sources, niacinamide etc)?Not bad man!
If you switch to Pitavastatin, you may see a 10+ point boost to HDL, a very clinically significant increase that would make plaque regression move at a good pace with your fairly low LDL. I saw an 11 point jump from 39 to 51 on 200mg Test. Unless you're on Atorvastatin due to kidney disease, Pita is superior.
If anyone needs inspiration, my lipids from September 2024:
To September 2025:
This is with Pitavastatin 4, Ezetimebe, and Repatha. The Repatha is only adding 10-12% more improvement to the other two though.
After these, and stress echo + CPET results were sent, my preventative cardiologist added a note:
"You have undergone a complete cardiometabolic risk inversion. From a 10-year ASCVD risk near 18%, you now present with optimal blood pressure, an LDL profile in the lowest 0.1% of the population, and endurance-level aerobic fitness. Your current risk is...
dinfar1337
New Member
your september cholesterol is my goal man.Not bad man!
If you switch to Pitavastatin, you may see a 10+ point boost to HDL, a very clinically significant increase that would make plaque regression move at a good pace with your fairly low LDL. I saw an 11 point jump from 39 to 51 on 200mg Test. Unless you're on Atorvastatin due to kidney disease, Pita is superior.
If anyone needs inspiration, my lipids from September 2024:
To September 2025:
This is with Pitavastatin 4, Ezetimebe, and Repatha. The Repatha is only adding 10-12% more improvement to the other two though.
After these, and stress echo + CPET results were sent, my preventative cardiologist added a note:
"You have undergone a complete cardiometabolic risk inversion. From a 10-year ASCVD risk near 18%, you now present with optimal blood pressure, an LDL profile in the lowest 0.1% of the population, and endurance-level aerobic fitness. Your current risk is...
im not on statins yet but with reta/cardio im on 45 hdl 60 ldl. trying to squeeze last bit of hdl up and ldl down before jumping on.
OldGHGuy
Member
Thanks brother. I will give Pita a try as moving the needle on HDL has been really tough. BTW - I bought some Ezitimebe on your recommendation but after these bloods I'm wondering if I need it? Thoughts?Not bad man!
If you switch to Pitavastatin, you may see a 10+ point boost to HDL, a very clinically significant increase that would make plaque regression move at a good pace with your fairly low LDL. I saw an 11 point jump from 39 to 51 on 200mg Test. Unless you're on Atorvastatin due to kidney disease, Pita is superior.
If anyone needs inspiration, my lipids from September 2024:
To September 2025:
This is with Pitavastatin 4, Ezetimebe, and Repatha. The Repatha is only adding 10-12% more improvement to the other two though.
After these, and stress echo + CPET results were sent, my preventative cardiologist added a note:
"You have undergone a complete cardiometabolic risk inversion. From a 10-year ASCVD risk near 18%, you now present with optimal blood pressure, an LDL profile in the lowest 0.1% of the population, and endurance-level aerobic fitness. Your current risk is...
Ghoul
Member
Ezetimibe is the most benign LDL reducing compound there is. Since it's "cost free", and there's a measurable benefit to getting LDL below 40 per the latest guidelines, I strongly recommend you use it. You're in or near plaque regression territory. Studies show lower LDL is the primary driver of regression. The sooner and faster regression takes place the "deeper" the cleaning of your arteries will be. The longer plaque remains the greater the amount that will harden and become unremovable,
The LDL drop from Ezetimebe isn't usually huge, but if you happen to be in the 15% of genetic "dietary cholesterol hyperabsorbers", it may be a very large reduction.
Ghoul
Member
None. The latest ESC (Euro Society of Cardiology) lipid guidelines directly addressed this. The problem with HDL increases from supplements is that they don't increase production of APO-I. This is the protein that gives HDL its ability to remove cholesterol from arteries and transport it back to the liver. So what's happening is you're producing low or non-functional HDL. At best it's neutral, but giving a deceptive sense of being useful based on the HDL number, At worst, there's evidence non functional HDL becomes inflammatory.
Pitavastatin stimulates the production of APO-I, along with HDL. So Pitavastatin is producing functional HDL. In fact the boost to APO-I is likely improving the function of all your HDL.
The only other way to significantly increase APO-I is exercise.
After LDL, HDL function (efflux capacity), is the greatest factor determining how much regression of plaque is possible.
The reason for the "rush" is that once LDL is low enough that you're no longer depositing it in arteries, there's a limited window for regression before plaque stabilizes, which is good, but means it's no longer able to be removed. Removal is better than stabilization.
You can get it tested with Boston Heart Diagnostics HDL Map test if you're curious, $169.
A new way of looking at ‘good’ cholesterol | Heart | UT Southwestern Medical Center
Knowing how your cholesterol functions can help doctors determine your heart disease risk. A UT Southwestern cardiologist uses a garbage truck analogy to explain “cholesterol efflux.”
utswmed.org
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