Boldenone (EQ) Misconceptions and Comparison with Metenolone (Primo) [Author: Type-IIx]

[Sector]

I really like all the thought you put into your plan and how it’s layed out. Very good approach.
I would drop the Tren for the bulk if you’re on the fence. Yes maximum anabolics is important and the role Tren plays is significant. But also consider the impact on health. Dropping the Tren in the bulk reduces the impact on your health and a healthy body is very important for growth and can outweigh the advantages Tren offers.
Im using Test/Tren in this cut. Have Deca in my system still, it’s working its way out, not injecting any. and it will be long out before this cut is over.
My Tren dose is low compared to my Test dose, will be tapering Test down also
Also using inj. Albuterol pre cardio
Really relying on my diet and training as the primacy drivers in this cut.

I like your plan with insulin post workout meal in your bulk, considering the same.
I’m relying on my low bodyfat and strict diet to maintain insulin sensitivity on high HGH won’t be using any insulin for this purpose. If my HGH is having an impact in this regard I’ll lower the dose, hopefully I won’t have to.
I like your detailed plan and will be looking forward to following your results and findings.
Good luck

Yeah likewise man! Great discussion!

How are you liking the albuterol pre workout? Never heard of injectable albuterol. Do you get the shakes from it like you would with clen?

 

[Deskoze38192]

Yeah likewise man! Great discussion!

How are you liking the albuterol pre workout? Never heard of injectable albuterol. Do you get the shakes from it like you would with clen?

I like it and mostly I like it because I can sleep fine. I mainly use it pre fasted cardio.
When I first started using it over a year ago I did get the shakes pretty bad. I ride my motorcycle to the gym most days so this was a factor lol. They pass quickly though. 10-20 mins after injection they are gone.
Now the shakes aren’t really noticeable even at a decent dose.

 

[eery]

Removing any influence of relative parent hormone content between the undecylenate & enanthate esters (where enanthate > undecylenate, i.e., Primo > EQ), we can say that boldenone is a surprisingly weak anabolic agent, with a low potency to activate the AR (10.72% relative potency vs. DHT). Metenolone is considerably more potent as an AR ligand (28.75% relative potency vs. DHT).

i thought boldenones binding to the AR was stronger than testosterone for some reason, where are these numbers from and do you know where i can read more about it? could you talk a bit more of how you think EQ compares to others anabolics when it comes to AR binding and tissue accrual?

I like it and mostly I like it because I can sleep fine. I mainly use it pre fasted cardio.
When I first started using it over a year ago I did get the shakes pretty bad. I ride my motorcycle to the gym most days so this was a factor lol. They pass quickly though. 10-20 mins after injection they are gone.
Now the shakes aren’t really noticeable even at a decent dose.

how much albuterol do you use when its injectable like that?

 

[Deskoze38192]

how much albuterol do you use when its injectable like that?

It’s a product sold by a very reliable source here.
I don’t believe he discloses the actual mcg amount of Albuterol for safety purposes because he doesn’t want people to take an equal amount to oral Clen because it would kill them, but rather I follow his recommendations for the amount used.
I think that’s the reason, someone that knows can correct me if I am wrong
And I believe it’s in mcg not Mg

 

[eery]

It’s a product sold by a very reliable source here.
I don’t believe he discloses the actual mcg amount of Albuterol for safety purposes because he doesn’t want people to take an equal amount to oral Clen because it would kill them, but rather I follow his recommendations for the amount used.
I think that’s the reason, someone that knows can correct me if I am wrong
And I believe it’s in mcg not Mg

sounds dangerous to buy a product you dont know the concentration on to me, i would want to know how much im taking.

 

[Deskoze38192]

sounds dangerous to buy a product you dont know the concentration on to me, i would want to know how much im taking.

Like I said I could be wrong. Just going off memory and my memory isn’t the best lol.
And you’re definitely make a very valid point but if you do the brewing math it’s not dangerous at all.
Let’s say the amount per mL in each 20 mL vial is 500 mcg. That’s 10 mg of product per vial. He’s not making one vial at a time so let’s say it’s 100 vials per batch. So he’s putting 1 gram of product in each batch.
Even if it’s off by 10% it’s not going to matter very much because it’s divided so many times it wouldn’t matter really.
Your only take 0.25 mL for your dose so the 1 gram in the batch is getting divided 500 times before you take your dose.
The problem lies in someone thinking they need to take much more than the recommended dose since the dose of Oral Clen is so much higher. The dose of Clen is actually what would be in 40 batches so absolutely not possible but the desire to take more is common in human nature
Also, I brew most everything I take if I knew the amount I for sure would get the raws and just make it mysell
Also I’ve used 140 mL so far without the slightest issue

Edit:I apologize to everyone for going way off the topic of EQ
Back to EQ…

 

[DonTesto]

This requires too many leaps of faith to state a claim either way (with respect to muscle anabolism).

What I can say is that I can make a case for Primo but not EQ use on the basis of anything tangible. Of course, this doesn't mean that if you like EQ, don't use it; and I do not address EQ's use for women, for whom it is particularly outstanding (perhaps indicating some intangible qualities that it possesses). So feel free to handwave, "meh, maybe on paper" away; but I will just defend this post by pointing out that it's not only typed out ("on paper") but also reasoned out.

Removing any influence of relative parent hormone content between the undecylenate & enanthate esters (where enanthate > undecylenate, i.e., Primo > EQ), we can say that boldenone is a surprisingly weak anabolic agent, with a low potency to activate the AR (10.72% relative potency vs. DHT). Metenolone is considerably more potent as an AR ligand (28.75% relative potency vs. DHT).

However, metenolone is principally metabolized by 3α-HSD (like DHT), ubiquitious in human skeletal muscle, meaning that it is basically broken down rapidly in muscle (in humans) before it is able to exert substantial muscle anabolism.

I can make a much stronger case for rational metenolone use vs. boldenone, as metenolone does pose somewhat reduced cardiac harm risks and is a commercially available (i.e., pharmaceutical) parenteral (i.m.) 5α-reduced AAS.

Boldenone basically does what nandrolone and testosterone do (augments IGF-I, increasing total-body size), but with risks attendant to veterinary & black market drug supplies, without offering any unique benefit. Unique benefits of testosterone include its 5α-reduction & supporting sexual function, and of nandrolone include its increasing matrix type I collagen deposition & fluid retention in synovial joints, benefiting joints acutely (but also posing unique cardiac harms).

Well, considering how EQ raises red blood cells (increased work capacity), can be used to lower estrogen, and how it is usually found at 300mg or even 500mg per mL for some 40-50 bucks. It has its uses vs primo which there is a shortage of lately and is often faked. EQ is more or less 6 times cheaper than primo.

 

[Type-IIx]

Well, considering how EQ raises red blood cells (increased work capacity), can be used to lower estrogen, and how it is usually found at 300mg or even 500mg per mL for some 40-50 bucks. It has its uses vs primo which there is a shortage of lately and is often faked. EQ is more or less 6 times cheaper than primo.

Supposing that EQ is most erythropoietic of all, and there's absolutely zero quality evidence of that, actually – besides plenty of marketing to stimulate sales for a product that doesn't sell much I understand – it's a bug not a feature to most, right? A top reason guys stop using certain gear like nandrolone – for which there is good evidence for its erythropoietic potency – is too high hematocrit.

Primo lowers estrogen better than EQ [and I was comparing the two], right?

Sure, you probably save a few bucks buying EQ… supply and demand.

EQ is my personal least favorite steroid that I've actually used/never used MENT but it's worse on paper.
–
All that being said, I use EQ with guys not that infrequently.

I can make decent arguments for using it but mostly on the basis of intangible qualities.

For example, it gives a pretty unique, hard to achieve otherwise, dry & full look. It's the aromatization mostly to E1 and quality mass it puts on, plus that vascularity from the RBC increase… had you said vascularity, I'd have agreed! It's not as good as nandrolone in that regard IMO, though, which I wrote about here: Nandrolone's unique aspects: vascularity

 

[DonTesto]

Supposing that EQ is most erythropoietic of all, and there's absolutely zero quality evidence of that, actually – besides plenty of marketing to stimulate sales for a product that doesn't sell much I understand – it's a bug not a feature to most, right? A top reason guys stop using certain gear like nandrolone – for which there is good evidence for its erythropoietic potency – is too high hematocrit.

Primo lowers estrogen better than EQ [and I was comparing the two], right?

Sure, you probably save a few bucks buying EQ… supply and demand.

EQ is my personal least favorite steroid that I've actually used/never used MENT but it's worse on paper.
–
All that being said, I use EQ with guys not that infrequently.

I can make decent arguments for using it but mostly on the basis of intangible qualities.

For example, it gives a pretty unique, hard to achieve otherwise, dry & full look. It's the aromatization mostly to E1 and quality mass it puts on, plus that vascularity from the RBC increase… had you said vascularity, I'd have agreed! It's not as good as nandrolone in that regard IMO, though, which I wrote about here: Nandrolone's unique aspects: vascularity

Intangible qualities? It does raise your red blood count better than most other roids, blood work of users generally show that. Same goes for the A.I properties. You can talk about the lack of studies all you want but for the purposes of this you really don't need anything other than people doing only test at X amount, doing blood work to check their e2, then adding EQ, without changing the dose of test, doing blood again and see the reduction of e2. That's pretty tangible buddy.

 

[Type-IIx]

Intangible qualities? It does raise your red blood count better than most other roids, blood work of users generally show that. Same goes for the A.I properties. You can talk about the lack of studies all you want but for the purposes of this you really don't need anything other than people doing only test at X amount, doing blood work to check their e2, then adding EQ, without changing the dose of test, doing blood again and see the reduction of e2. That's pretty tangible buddy.

More is not better, cheaper isn't better; lack of studies means without hard evidence to meeee but ya whatever you say bud!

 

[koast123]

Supposing that EQ is most erythropoietic of all, and there's absolutely zero quality evidence of that, actually – besides plenty of marketing to stimulate sales for a product that doesn't sell much I understand – it's a bug not a feature to most, right? A top reason guys stop using certain gear like nandrolone – for which there is good evidence for its erythropoietic potency – is too high hematocrit.

Primo lowers estrogen better than EQ [and I was comparing the two], right?

Sure, you probably save a few bucks buying EQ… supply and demand.

EQ is my personal least favorite steroid that I've actually used/never used MENT but it's worse on paper.
–
All that being said, I use EQ with guys not that infrequently.

I can make decent arguments for using it but mostly on the basis of intangible qualities.

For example, it gives a pretty unique, hard to achieve otherwise, dry & full look. It's the aromatization mostly to E1 and quality mass it puts on, plus that vascularity from the RBC increase… had you said vascularity, I'd have agreed! It's not as good as nandrolone in that regard IMO, though, which I wrote about here: Nandrolone's unique aspects: vascularity

Whats your favourites steroids?

 

[xrayphoton13]

Whats your favourites steroids?

Post in thread 'Your Top 5 Fav Steroids' Your Top 5 Fav Steroids

 

[koast123]

Post in thread 'Your Top 5 Fav Steroids' Your Top 5 Fav Steroids

Oh thanks, my fault, i didnt know that post. u very kind!

 

[Type-IIx]

Whats your favourites steroids?

Sup koast bro, long time!


My favorites are like @xrayphoton13 linked, he's a weapon at finding these posts. Some like Anadrol are more sentimental, I just like it cause I do well on it and like the strength effects. I get a lot of strength from Var, too.

Practically I believe that anyone who really groks the Anabolic Insights: Unique Characteristics of Anabolic-Androgenic Steroids series can learn all there is to know about cycle design.

The next couple articles coming out for Meso and my book Ampoule: A Guide to Anabolic Cycle Design which I've been writing (and published some of the first chapter from on Substack/to founding members) will give the framework, drill home some points and show things like edge cases, too

 

[Photon]

1. Misconception that boldenone is nonaromatizable, or that its use (in sufficiently high doses) permit abstaining from AI/SERM use in users that are not resilient against aromatizing effects (e.g., fluid retention/edema, growth of breast tissue, etc.)

Reality: Boldenone aromatizes (to estrone [E1] & estradiol [E2]) at 58% the rate of testosterone. This is significant. However, since Estrone (E1) is a weak estrogen, because lacks the 17β-OH group, it possesses only 2% of E2's potency to transactivate ERα (associated with aromatizing effects, many of which are negative side effects in men).

Practically, this means that moderate/moderately-high boldenone doses (used solo; or in combination with nonaromatizable drugs, and/or with low doses of exogenous testosterone) would tend to not require AI/SERM use in users that are not resilient against aromatizing effects, but: A) for those who principally aromatize to E2 rather than E1 (inter-individual differences are likely rooted in 17β-HSD1 isozyme expression, under the control of the A1-Q327 gene) particular; and/or B) where E1 blood concentrations start getting into the regions of > 50-fold above that at which E2 causes problems in the individual, then the aromatizing effects would be expected to predominate.

Have you done labwork for E1 on your clients?
I have my labs posted here with 1g T and 1g EQ with no AI.
It looks like it crashes my E2 and leaves me with low E1. I would have expected E1 to be much higher.

Steroid Profile Post in thread 'Equipoise'

Oct 15, 2025

highly likely eq outcompeting test at the aromatese receptor. can speed slowed aromatese up by adding dianabol in or upping testosterone.

you would probably need to lower eq or up test. or run higher dbol to outcompete the eq at receptor site level. this is inhibition of aromatese, not e2>e1 conversion problem

Already on 1g TD / 1g EQ lol
e2 standalone test


4 weeks later, with e2 injected
e1+e2 test



I'm considering 20dbol a day vs injecting e2.

• Photon

• 

 

[Type-IIx]

Have you done labwork for E1 on your clients?
I have my labs posted here with 1g T and 1g EQ with no AI.
It looks like it crashes my E2 and leaves me with low E1. I would have expected E1 to be much higher.

Steroid Profile Post in thread 'Equipoise'

Oct 15, 2025

highly likely eq outcompeting test at the aromatese receptor. can speed slowed aromatese up by adding dianabol in or upping testosterone.

you would probably need to lower eq or up test. or run higher dbol to outcompete the eq at receptor site level. this is inhibition of aromatese, not e2>e1 conversion problem

Already on 1g TD / 1g EQ lol
e2 standalone test


4 weeks later, with e2 injected
e1+e2 test



I'm considering 20dbol a day vs injecting e2.

• Photon

• 

That’s so unusual, NEVER seen this attributed to EQ. What else were you on? AI? Etc.

You’d have to give your records for the 6+ weeks prior for everything that affects estrogens, things like androgens and AIs of course.

 

[Type-IIx]

And if you haven’t read it, I recommend Primobolan / Equipoise Crashed my E2 – Help! on this topic

 

[Type-IIx]

Also, I’m not going to analyze your bloodwork, but just so you understand that you can’t claim EQ had this effect without charting everything out for the 6 weeks prior to the blood draw.

 

[Type-IIx]

This is what happens if you take your bloodwork soon after starting EQ though! You want to wait until it hits steady-state and it’s a long ester. Depending on an individual’s binding hormone profile, the slower release from depot for EQ before reaching steady-state will almost certainly result in reduced Aromatase activity.

 

[Photon]

That’s so unusual, NEVER seen this attributed to EQ. What else were you on? AI? Etc.

You’d have to give your records for the 6+ weeks prior for everything that affects estrogens, things like androgens and AIs of course.

End of blast.
Close to week 20?
1g test d
1g eq
80mg trestolone deconate

TNE/mtren pwo (g-free)
40mg halo
Rotated between adrol n var

No AI

Started running e2 cyp as I went off trestolone.