Anyone have experiences with test undecanoate?

[Spaceman Spiff]

@Banana Joe
You cant even read a line graph properly..

How the fuck is the top line chat show signs of more stability than the bottom one? The saw tooth means it has higher peaks and troughs...

 

[Banana Joe]

I am the biggest .... of ... here
... I have contributed more than anyone else here ...

Any post like that rarely proves to be of true substance in an internet forum, but let's have a closer look at it...

@Banana Joe
You cant even read a line graph properly..

Apparently you can't read anything properly.
Please explain to me what data of yours is RELEVANT to answering the question, where my gyno flare up came from. Your last two graphs simply show that you are unable to comprehend what you are reading.
This obvious inability make you argue your own strawman, presumably out of a lack of intellect and not out of disingenuous intent, not a claim that I have made. Never ever did I claim that E provides the same stable graph as U, when both are injected at the same interval.
Anyone with just basic level of knowledge would know that, congratulations for being one of them.

Test U is 100% more stable....

No, TU is not more stable than TU. You are provably unable to comprehend even just simple contexts.

look here dipshit.

Ok, let's play that game, where you insult me with cocky confidence, because you got all the facts on your side. I can see that you randomly quoted yourself here, but I am asking once again:

WHERE is the graph that shows one has significantly higher E2 peaks, when taking 250mg of TU, compared to 500mg of TE - both with appropriate front loading according to ALL of those calculators, and then using appropriate intervals to maintain stable levels of total T in blood serum? Note that I used "appropriate" here, do you understand that word in that context?

Next time, try quoting specifically the false claims that I made, or where my reasoning is based on false assumptions, with the corresponding rebuttal, but not full quotes of yourself about things nobody but you is talking about - or my full post, with just the addition of "your wrong bro".

Feel to prove me wrong, I got no ego on such things. I am just annoyed as fuck with people like you, that are unable to read, let alone comprehend and then think they got a point to make, having no base to their "argument" but lots of petty insults to throw around.

I know nothing of that message will reach or get to you, as you are a prime example for a person with low ability or knowledge in a specific area, that overestimates its own competence, often significantly overrating its skills relative to objective measures and to others - aka the Dunning-Kruger-effect.

Unless you got something substantial to say, this will be my last reply to your posts.

Have a nice day, buddy!

 

[Spaceman Spiff]

Any post like that rarely proves to be of true substance in an internet forum, but let's have a closer look at it...


Apparently you can't read anything properly.
Please explain to me what data of yours is RELEVANT to answering the question, where my gyno flare up came from. Your last two graphs simply show that you are unable to comprehend what you are reading.
This obvious inability make you argue your own strawman, presumably out of a lack of intellect and not out of disingenuous intent, not a claim that I have made. Never ever did I claim that E provides the same stable graph as U, when both are injected at the same interval.
Anyone with just basic level of knowledge would know that, congratulations for being one of them.


No, TU is not more stable than TU. You are provably unable to comprehend even just simple contexts.


Ok, let's play that game, where you insult me with cocky confidence, because you got all the facts on your side. I can see that you randomly quoted yourself here, but I am asking once again:

WHERE is the graph that shows one has significantly higher E2 peaks, when taking 250mg of TU, compared to 500mg of TE - both with appropriate front loading according to ALL of those calculators, and then using appropriate intervals to maintain stable levels of total T in blood serum? Note that I used "appropriate" here, do you understand that word in that context?

Next time, try quoting specifically the false claims that I made, or where my reasoning is based on false assumptions, with the corresponding rebuttal, but not full quotes of yourself about things nobody but you is talking about - or my full post, with just the addition of "your wrong bro".

Feel to prove me wrong, I got no ego on such things. I am just annoyed as fuck with people like you, that are unable to read, let alone comprehend and then think they got a point to make, having no base to their "argument" but lots of petty insults to throw around.

I know nothing of that message will reach or get to you, as you are a prime example for a person with low ability or knowledge in a specific area, that overestimates its own competence, often significantly overrating its skills relative to objective measures and to others - aka the Dunning-Kruger-effect.

Unless you got something substantial to say, this will be my last reply to your posts.

Have a nice day, buddy!

Think what you want. You havent put in any data here for test U, no blood work. Nothing.

Find someone else who has discussed it more than I have on the forum, or have posted different topics of even blood work reporting. There is little to nothing

You cannot say it causes higher peaks of e2 because you have no data and literally went off of feels. You are just throwing a bunch of hypotheticals.

You really are a pathetic son of a bitch

 

[Banana Joe]

Find someone else who has discussed it more than I have on the forum, or have posted different topics of even blood work reporting. There is little to nothing

That is not my obligation here. You made a claim, therefore you have to show proof, or at least provide some reasoning why you have come to such a bold conclusion.

Even if everything I reason or assume is wrong - we will not be able to exchange arguments, or have a meaningful discussion for that matter, as you will be entirely unable to understand what I am stating, or at least trying to convey.
It is sadly that simple.

Your clear inability to understand what is written makes me feel sorry for you. And believe it or not, I am not saying this to be condescending.

You really are a pathetic son of a bitch

Q.E.D.


Like I said, there is no discussion to be had with you, therefore I will now end it on my side. Feel free to keep arguing your own strawman or throw insults at me.

 

[dinfar1337]

You are just playing the guessing game. You have no data dude...

i've been trying to explain to this dude you can offset a 2g frontload in 21 days instead of the traditionally frontload on test enethate on 3.5 days. so while he got stable with TE in less than a week his estrogen was likely elevated for too long with testosterone U which caused gyno.

which is a guess you're completely right

have no bloods, no carrier oil excipents, no dosaging ran by x week. just a bunch of headache

 

[dinfar1337]

How is E2 level dependant on the ester attached to the test and and not total test in my blood?

because the ester releases testosterone into your blood at an x spefiic rate!

you cant just bulk 50kg in one day and expect to gain 50kg of muscle mass

 

[verylargeshoes]

Main things - it aromatizes more than other esters. So if you're already a heavy aromatizer then it might not be a great option for TRT.

@drbeefsupreme can you talk about this a bit more? When you say it "aromatizes more", are you saying that a given mass of testosterone cleaved from the U ester would convert to more estrogen than the same mass cleaved from cyp or enanthate, or is this more a statement about differing pharmacokinetics, or perhaps something to do with common administration protocols, or ...?

 

[dinfar1337]

@drbeefsupreme can you talk about this a bit more? When you say it "aromatizes more", are you saying that a given mass of testosterone cleaved from the U ester would convert to more estrogen than the same mass cleaved from cyp or enanthate, or is this more a statement about differing pharmacokinetics, or perhaps something to do with common administration protocols, or ...?

this is totally bullshit, you get testosterone absorbed into your body, not the ester when its gone.

if there is any different its not significant within 0.01pg estrongen if its from the ester. and thats being generous

 

[verylargeshoes]

this is totally bullshit, you get testosterone absorbed into your body, not the ester when its gone.

if there is any different its not significant within 0.01pg estrongen if its from the ester. and thats being generous

It's interesting -- lots of people talk about perceived differences in behavior from the different esters. Some of them may simply be functions of differing rates of production of available testosterone vs. the body's bandwidth for aromatization, or differences in administration (e.g. test U gets frontloaded, which is a pretty big variable) that may produce effects not captured in the simple models we use. Some of them may just be unreliable data. There's a heck of a lot of it, though.

For example, very experienced people say things like "NPP doesn't help with joint pain as much as Deca, and I never get Deca Dick with NPP". Or "I don't get high estrogen or sides from using high doses of TNE several times a week".

I'm just a simple country lawyer, and I don't understand any of these processes well enough to make intelligent guesses.

 

[dinfar1337]

It's interesting -- lots of people talk about perceived differences in behavior from the different esters. Some of them may simply be functions of differing rates of production of available testosterone vs. the body's bandwidth for aromatization, or differences in administration (e.g. test U gets frontloaded, which is a pretty big variable) that may produce effects not captured in the simple models we use. Some of them may just be unreliable data. There's a heck of a lot of it, though.

For example, very experienced people say things like "NPP doesn't help with joint pain as much as Deca, and I never get Deca Dick with NPP". Or "I don't get high estrogen or sides from using high doses of TNE several times a week".

I'm just a simple country lawyer, and I don't understand any of these processes well enough to make intelligent guesses.

you are on the right track with timeline of the ester, and that has nothing to do with how you aromatize.

testosterone is testosterone

500mg will aromatize the same as 500mg. but you are never gonna get 500mg of ester x == ester y, because theres so many different variables with esters.

 

[verylargeshoes]

but you are never gonna get 500mg of ester x == ester y, because theres so many different variables with esters.

Ester weight variation is something I hope people are already taking into account. What are the other variables?

 

[dinfar1337]

Ester weight variation is something I hope people are already taking into account. What are the other variables?

its different from ester to ester

but generally chain length ( timeline for ester to release test, example 50mg x day. the longer the the chain the less test goes through a day and the shorter it is the more goes through each day ),

half life we already talked about,

how you metabolise the ester in your liver which is upto how healthy you are
and your genetics ( better liver more grams you can blast woohoo)

which carrier oil is used? and how quickly do you absorb it


this above is what mostly affects aromatizion, otherwise test is test and will mostly be aromatized the same.

oh and i can tell you Test U has the biggest chain length of any ester we widely use for testosterone. so not only does it have the longest half life, it also is being the most slowly released per day of any steroids